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The Nidoviruses: Toward Control of SARS and other Nidovirus Diseases

Stanley Perlman ; Kathryn V. Holmes (eds.)

Resumen/Descripción – provisto por la editorial

No disponible.

Palabras clave – provistas por la editorial

Immunology; Infectious Diseases; Microbiology; Epidemiology; Virology; Pathology

Disponibilidad
Institución detectada Año de publicación Navegá Descargá Solicitá
No detectada 2006 SpringerLink

Información

Tipo de recurso:

libros

ISBN impreso

978-0-387-26202-4

ISBN electrónico

978-0-387-33012-9

Editor responsable

Springer Nature

País de edición

Reino Unido

Fecha de publicación

Información sobre derechos de publicación

© Springer US 2006

Tabla de contenidos

Efficacy of an Inactivated PRRSV Vaccine: Induction of Virus-Neutralizing Antibodies and Partial Virological Protection upon Challenge

Gerald Misinzo; Peter L. Delputte; Peter Meerts; Christa Drexler; Hans J. Nauwynck

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

VI - Pathogenesis of Arteriviruses and Toroviruses | Pp. 449-454

Sars and Other Coronaviruses in Humans and Animals

Leo L. M. Poon

In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.

VII - Pathogenesis of Human Coronaviruses | Pp. 457-462

Animal Models for Sars

Anjeanette Roberts; Kanta Subbarao

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

VII - Pathogenesis of Human Coronaviruses | Pp. 463-471

HCoV-Oc43–Induced Apoptosis of Murine Neuronal Cells

Hélène Jacomy; Pierre J. Talbot

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

VII - Pathogenesis of Human Coronaviruses | Pp. 473-478

Infection of Human Airway Epithelia by Sars Coronavirus is Associated with ACE2 Expression and Localization

Hong Peng Jia; Dwight C. Look; Melissa Hickey; Lei Shi; Lecia Pewe; Jason Netland; Michael Farzan; Christine Wohlford-Lenane; Stanley Perlman; Paul B. McCray

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

VII - Pathogenesis of Human Coronaviruses | Pp. 479-484

Human Coronavirus NL63 Infection is Associated with Croup

Alexander Stang; Krzysztof Pyrc; Maarten F. Jebbink; Gudula Petersen; Johannes Forster; Ben Berkhout; Klaus Überla; Lia van der Hoek; Klaus Sure; Gabriele Ihorst

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

VII - Pathogenesis of Human Coronaviruses | Pp. 485-491

A SARS-CoV–Specific Protein Enhances Virulence of an Attenuated Strain of Mouse Hepatitis Virus

Lecia Pewe; Haixia Zhou; Jason Netland; Chandra Tangadu; Heidi Olivares; Lei Shi; Dwight Look; Thomas Gallagher; Stanley Perlman

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

VII - Pathogenesis of Human Coronaviruses | Pp. 493-498

Genetic Evolution of Human Coronavirus OC43 in Neural Cell Culture

Julien R. St-Jean; Marc Desforges; Pierre J. Talbot

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

VII - Pathogenesis of Human Coronaviruses | Pp. 499-502

Synergistic Inhibition of Sars-Coronavirus Replication by Type I and Type II IFN

Eric C. Mossel; Bruno Sainz; Robert F. Garry; C. J. Peters

In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.

VII - Pathogenesis of Human Coronaviruses | Pp. 503-506

ACE2 Functions as a Receptor for Sars-Coronavirus

Lindsay K. Heller; Laura Gillim-Ross; Emily R. Olivieri; David E. Wentworth

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

VII - Pathogenesis of Human Coronaviruses | Pp. 507-510