Catálogo de publicaciones - libros
The Nidoviruses: Toward Control of SARS and other Nidovirus Diseases
Stanley Perlman ; Kathryn V. Holmes (eds.)
Resumen/Descripción – provisto por la editorial
No disponible.
Palabras clave – provistas por la editorial
Immunology; Infectious Diseases; Microbiology; Epidemiology; Virology; Pathology
Disponibilidad
Institución detectada | Año de publicación | Navegá | Descargá | Solicitá |
---|---|---|---|---|
No detectada | 2006 | SpringerLink |
Información
Tipo de recurso:
libros
ISBN impreso
978-0-387-26202-4
ISBN electrónico
978-0-387-33012-9
Editor responsable
Springer Nature
País de edición
Reino Unido
Fecha de publicación
2006
Información sobre derechos de publicación
© Springer US 2006
Cobertura temática
Tabla de contenidos
Efficacy of an Inactivated PRRSV Vaccine: Induction of Virus-Neutralizing Antibodies and Partial Virological Protection upon Challenge
Gerald Misinzo; Peter L. Delputte; Peter Meerts; Christa Drexler; Hans J. Nauwynck
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
VI - Pathogenesis of Arteriviruses and Toroviruses | Pp. 449-454
Sars and Other Coronaviruses in Humans and Animals
Leo L. M. Poon
In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.
VII - Pathogenesis of Human Coronaviruses | Pp. 457-462
Animal Models for Sars
Anjeanette Roberts; Kanta Subbarao
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
VII - Pathogenesis of Human Coronaviruses | Pp. 463-471
HCoV-Oc43–Induced Apoptosis of Murine Neuronal Cells
Hélène Jacomy; Pierre J. Talbot
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
VII - Pathogenesis of Human Coronaviruses | Pp. 473-478
Infection of Human Airway Epithelia by Sars Coronavirus is Associated with ACE2 Expression and Localization
Hong Peng Jia; Dwight C. Look; Melissa Hickey; Lei Shi; Lecia Pewe; Jason Netland; Michael Farzan; Christine Wohlford-Lenane; Stanley Perlman; Paul B. McCray
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
VII - Pathogenesis of Human Coronaviruses | Pp. 479-484
Human Coronavirus NL63 Infection is Associated with Croup
Alexander Stang; Krzysztof Pyrc; Maarten F. Jebbink; Gudula Petersen; Johannes Forster; Ben Berkhout; Klaus Überla; Lia van der Hoek; Klaus Sure; Gabriele Ihorst
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
VII - Pathogenesis of Human Coronaviruses | Pp. 485-491
A SARS-CoV–Specific Protein Enhances Virulence of an Attenuated Strain of Mouse Hepatitis Virus
Lecia Pewe; Haixia Zhou; Jason Netland; Chandra Tangadu; Heidi Olivares; Lei Shi; Dwight Look; Thomas Gallagher; Stanley Perlman
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
VII - Pathogenesis of Human Coronaviruses | Pp. 493-498
Genetic Evolution of Human Coronavirus OC43 in Neural Cell Culture
Julien R. St-Jean; Marc Desforges; Pierre J. Talbot
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
VII - Pathogenesis of Human Coronaviruses | Pp. 499-502
Synergistic Inhibition of Sars-Coronavirus Replication by Type I and Type II IFN
Eric C. Mossel; Bruno Sainz; Robert F. Garry; C. J. Peters
In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.
VII - Pathogenesis of Human Coronaviruses | Pp. 503-506
ACE2 Functions as a Receptor for Sars-Coronavirus
Lindsay K. Heller; Laura Gillim-Ross; Emily R. Olivieri; David E. Wentworth
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
VII - Pathogenesis of Human Coronaviruses | Pp. 507-510