Catálogo de publicaciones - libros
The Nidoviruses: Toward Control of SARS and other Nidovirus Diseases
Stanley Perlman ; Kathryn V. Holmes (eds.)
Resumen/Descripción – provisto por la editorial
No disponible.
Palabras clave – provistas por la editorial
Immunology; Infectious Diseases; Microbiology; Epidemiology; Virology; Pathology
Disponibilidad
Institución detectada | Año de publicación | Navegá | Descargá | Solicitá |
---|---|---|---|---|
No detectada | 2006 | SpringerLink |
Información
Tipo de recurso:
libros
ISBN impreso
978-0-387-26202-4
ISBN electrónico
978-0-387-33012-9
Editor responsable
Springer Nature
País de edición
Reino Unido
Fecha de publicación
2006
Información sobre derechos de publicación
© Springer US 2006
Cobertura temática
Tabla de contenidos
Porcine Arterivirus Entry in Macrophages: Heparan Sulfate – Mediated Attachment, Sialoadhesin-Mediated Internalization, and a Cell-Specific Factor Mediating Virus Disassembly and Genome Release
Peter L. Delputte; H. J. Nauwynck
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 247-252
Enhancement of SARS-CoV Infection by Proteases
Shutoku Matsuyama; Makoto Ujike; Koji Ishii; Shuetsu Fukushi; Shigeru Morikawa; Masato Tashiro; Fumihiro Taguchi
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 253-258
Increased Viral Titers and Subtle Changes in Plaque Morphology Upon Passage of SARS-CoV in Cells from Different Species
Laura Gillim-Ross; Lindsay K. Heller; Emily R. Olivieri; David E. Wentworth
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 259-263
Human Coronavirus 229E can Use CD209L (L-Sign) to Enter Cells
Scott A. Jeffers; Erin M. Hemmila; Kathryn V. Holmes
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 265-269
Intracellular Transport of the S Proteins of Coronaviruses
Christel Schwegmann-Weβels; Xiaofeng Ren; Georg Herrler
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 271-275
Analysis of SARS-CoV Receptor Activity of ACE2 Orthologs
Emily R. Olivieri; Lindsey K. Heller; Laura Gillim-Ross; David E. Wentworth
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 277-280
Interaction Between the Spike Protein of Human Coronavirus NL63 and its Cellular Receptor ACE2
Stefan Pöhlmann; Thomas Gramberg; Anja Wegele; Krzysztof Pyrc; Lia van der Hoek; Ben Berkhout; Heike Hofmann
In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.
IV - Viral Entry | Pp. 281-284
Human Angiotensin - Converting Enzyme 2 (ACE2) is a Receptor for Human Respiratory Coronavirus NL63
M. K. Smith; Sonia Tusell; Emily A. Travanty; Ben Berkhout; Lia van der Hoek; Kathryn V. Holmes
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 285-288
Molecular Interactions of Group 1 Coronaviruses with Feline APN
Sonia M. Tusell; Kathryn V. Holmes
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 289-291
Pseudotyped Vesicular Stomatitis Virus for Functional Analysis of Sars Coronavirus Spike Protein
Shuetsu Fukushi; Tetsuya Mizutani; Masayuki Saijo; Shutoku Matsuyama; Fumihiro Taguchi; Ichiro Kurane; Shigeru Morikawa
In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.
IV - Viral Entry | Pp. 293-296