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The Nidoviruses: Toward Control of SARS and other Nidovirus Diseases

Stanley Perlman ; Kathryn V. Holmes (eds.)

Resumen/Descripción – provisto por la editorial

No disponible.

Palabras clave – provistas por la editorial

Immunology; Infectious Diseases; Microbiology; Epidemiology; Virology; Pathology

Disponibilidad
Institución detectada Año de publicación Navegá Descargá Solicitá
No detectada 2006 SpringerLink

Información

Tipo de recurso:

libros

ISBN impreso

978-0-387-26202-4

ISBN electrónico

978-0-387-33012-9

Editor responsable

Springer Nature

País de edición

Reino Unido

Fecha de publicación

Información sobre derechos de publicación

© Springer US 2006

Tabla de contenidos

Porcine Arterivirus Entry in Macrophages: Heparan Sulfate – Mediated Attachment, Sialoadhesin-Mediated Internalization, and a Cell-Specific Factor Mediating Virus Disassembly and Genome Release

Peter L. Delputte; H. J. Nauwynck

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 247-252

Enhancement of SARS-CoV Infection by Proteases

Shutoku Matsuyama; Makoto Ujike; Koji Ishii; Shuetsu Fukushi; Shigeru Morikawa; Masato Tashiro; Fumihiro Taguchi

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 253-258

Increased Viral Titers and Subtle Changes in Plaque Morphology Upon Passage of SARS-CoV in Cells from Different Species

Laura Gillim-Ross; Lindsay K. Heller; Emily R. Olivieri; David E. Wentworth

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 259-263

Human Coronavirus 229E can Use CD209L (L-Sign) to Enter Cells

Scott A. Jeffers; Erin M. Hemmila; Kathryn V. Holmes

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 265-269

Intracellular Transport of the S Proteins of Coronaviruses

Christel Schwegmann-Weβels; Xiaofeng Ren; Georg Herrler

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 271-275

Analysis of SARS-CoV Receptor Activity of ACE2 Orthologs

Emily R. Olivieri; Lindsey K. Heller; Laura Gillim-Ross; David E. Wentworth

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 277-280

Interaction Between the Spike Protein of Human Coronavirus NL63 and its Cellular Receptor ACE2

Stefan Pöhlmann; Thomas Gramberg; Anja Wegele; Krzysztof Pyrc; Lia van der Hoek; Ben Berkhout; Heike Hofmann

In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.

IV - Viral Entry | Pp. 281-284

Human Angiotensin - Converting Enzyme 2 (ACE2) is a Receptor for Human Respiratory Coronavirus NL63

M. K. Smith; Sonia Tusell; Emily A. Travanty; Ben Berkhout; Lia van der Hoek; Kathryn V. Holmes

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 285-288

Molecular Interactions of Group 1 Coronaviruses with Feline APN

Sonia M. Tusell; Kathryn V. Holmes

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 289-291

Pseudotyped Vesicular Stomatitis Virus for Functional Analysis of Sars Coronavirus Spike Protein

Shuetsu Fukushi; Tetsuya Mizutani; Masayuki Saijo; Shutoku Matsuyama; Fumihiro Taguchi; Ichiro Kurane; Shigeru Morikawa

In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.

IV - Viral Entry | Pp. 293-296