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The Nidoviruses: Toward Control of SARS and other Nidovirus Diseases

Stanley Perlman ; Kathryn V. Holmes (eds.)

Resumen/Descripción – provisto por la editorial

No disponible.

Palabras clave – provistas por la editorial

Immunology; Infectious Diseases; Microbiology; Epidemiology; Virology; Pathology

Disponibilidad
Institución detectada Año de publicación Navegá Descargá Solicitá
No detectada 2006 SpringerLink

Información

Tipo de recurso:

libros

ISBN impreso

978-0-387-26202-4

ISBN electrónico

978-0-387-33012-9

Editor responsable

Springer Nature

País de edición

Reino Unido

Fecha de publicación

Información sobre derechos de publicación

© Springer US 2006

Tabla de contenidos

Subcellular Localization of SARS-CoV Structural Proteins

Lisa A. Lopez; Ariel Jones; William D. Arndt; Brenda G. Hogue

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 297-300

Spike Gene Determinants of Mouse Hepatitis Virus Host Range Expansion

Willie C. McRoy; Ralph S. Baric

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 301-304

Virion-Liposome Interactions Identify a Cholesterol-Independent Coronavirus Entry Stage

Joseph A. Boscarino; Jeffrey M. Goletz; Thomas M. Gallagher

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 305-308

Avian Infectious Bronchitis Virus Enters Cells Via the Endocytic Pathway

Victor C. Chu; Lisa J. McElroy; A Damon Ferguson; Beverley E. Bauman; Gary R. Whittaker

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 309-312

Three-Dimensional Reconstruction of the Nucleolus Using Meta-Confocal Microscopy in Cells Expressing the Coronavirus Nucleoprotein

Jae-Hwan You; Mark L. Reed; Brian K. Dove; Julian A. Hiscox

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 313-318

Dissection of the Fusion Machine of Sars-Coronavirus

Megan W. Howard; Brian Tripet; Michael G. Jobling; Randall K. Holmes; Kathryn V. Holmes; Robert S. Hodges

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 319-322

Characterization of Persistent SARS-CoV Infection in Vero E6 Cells

Tetsuya Mizutani; Shuetsu Fukushi; Masayuki Saijo; Ichiro Kurane; Shigeru Morikawa

In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.

IV - Viral Entry | Pp. 323-326

Receptor-Independent Spread of a Neurotropic Murine Coronavirus MHV-JHMv in Mixed Neural Culture

Keiko Nakagaki; Kazuhide Nakagaki; Fumihiro Taguchi

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 327-330

Receptor-Independent Infection of Mouse Hepatitis Virus: Analysis by Spinoculation

Rie Watanabe; Kazumitsu Suzuki; Fumihiro Taguchi

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 331-334

SARS-CoV, But not HCoV-NL63, Utilizes Cathepsins to Infect Cells: Viral Entry

I-Chueh Huang; Berend Jan Bosch; Wenhui Li; Michael Farzan; Peter M. Rottier; Hyeryun Choe

In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.

IV - Viral Entry | Pp. 335-338