Catálogo de publicaciones - libros
The Nidoviruses: Toward Control of SARS and other Nidovirus Diseases
Stanley Perlman ; Kathryn V. Holmes (eds.)
Resumen/Descripción – provisto por la editorial
No disponible.
Palabras clave – provistas por la editorial
Immunology; Infectious Diseases; Microbiology; Epidemiology; Virology; Pathology
Disponibilidad
Institución detectada | Año de publicación | Navegá | Descargá | Solicitá |
---|---|---|---|---|
No detectada | 2006 | SpringerLink |
Información
Tipo de recurso:
libros
ISBN impreso
978-0-387-26202-4
ISBN electrónico
978-0-387-33012-9
Editor responsable
Springer Nature
País de edición
Reino Unido
Fecha de publicación
2006
Información sobre derechos de publicación
© Springer US 2006
Cobertura temática
Tabla de contenidos
Subcellular Localization of SARS-CoV Structural Proteins
Lisa A. Lopez; Ariel Jones; William D. Arndt; Brenda G. Hogue
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 297-300
Spike Gene Determinants of Mouse Hepatitis Virus Host Range Expansion
Willie C. McRoy; Ralph S. Baric
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 301-304
Virion-Liposome Interactions Identify a Cholesterol-Independent Coronavirus Entry Stage
Joseph A. Boscarino; Jeffrey M. Goletz; Thomas M. Gallagher
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 305-308
Avian Infectious Bronchitis Virus Enters Cells Via the Endocytic Pathway
Victor C. Chu; Lisa J. McElroy; A Damon Ferguson; Beverley E. Bauman; Gary R. Whittaker
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 309-312
Three-Dimensional Reconstruction of the Nucleolus Using Meta-Confocal Microscopy in Cells Expressing the Coronavirus Nucleoprotein
Jae-Hwan You; Mark L. Reed; Brian K. Dove; Julian A. Hiscox
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 313-318
Dissection of the Fusion Machine of Sars-Coronavirus
Megan W. Howard; Brian Tripet; Michael G. Jobling; Randall K. Holmes; Kathryn V. Holmes; Robert S. Hodges
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 319-322
Characterization of Persistent SARS-CoV Infection in Vero E6 Cells
Tetsuya Mizutani; Shuetsu Fukushi; Masayuki Saijo; Ichiro Kurane; Shigeru Morikawa
In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.
IV - Viral Entry | Pp. 323-326
Receptor-Independent Spread of a Neurotropic Murine Coronavirus MHV-JHMv in Mixed Neural Culture
Keiko Nakagaki; Kazuhide Nakagaki; Fumihiro Taguchi
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 327-330
Receptor-Independent Infection of Mouse Hepatitis Virus: Analysis by Spinoculation
Rie Watanabe; Kazumitsu Suzuki; Fumihiro Taguchi
So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.
IV - Viral Entry | Pp. 331-334
SARS-CoV, But not HCoV-NL63, Utilizes Cathepsins to Infect Cells: Viral Entry
I-Chueh Huang; Berend Jan Bosch; Wenhui Li; Michael Farzan; Peter M. Rottier; Hyeryun Choe
In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.
IV - Viral Entry | Pp. 335-338