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The Nidoviruses: Toward Control of SARS and other Nidovirus Diseases

Stanley Perlman ; Kathryn V. Holmes (eds.)

Resumen/Descripción – provisto por la editorial

No disponible.

Palabras clave – provistas por la editorial

Immunology; Infectious Diseases; Microbiology; Epidemiology; Virology; Pathology

Disponibilidad
Institución detectada Año de publicación Navegá Descargá Solicitá
No detectada 2006 SpringerLink

Información

Tipo de recurso:

libros

ISBN impreso

978-0-387-26202-4

ISBN electrónico

978-0-387-33012-9

Editor responsable

Springer Nature

País de edición

Reino Unido

Fecha de publicación

Información sobre derechos de publicación

© Springer US 2006

Tabla de contenidos

Ultrastructure of SARS-CoV, FIPV, and MHV Revealed by Electron Cryomicroscopy

Benjamin W. Neuman; Brian D. Adair; Craig Yoshioka; Joel D. Quispe; Ronald A. Milligan; Mark Yeager; Michael J. Buchmeier

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

III - Viral Assembly and Release | Pp. 181-185

Role of Mouse Hepatitis Coronavirus Envelope Protein Transmembrane Domain

Ye Ye; Brenda G. Hogue

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

III - Viral Assembly and Release | Pp. 187-191

The Transmembrane Domain of the Infectious Bronchitis Virus E Protein is Required for Efficient Virus Release

Carolyn E. Machamer; Soonjeon Youn

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

III - Viral Assembly and Release | Pp. 193-198

Viroporin Activity of SARS-CoV E Protein

Ying Liao; James P. Tam; Ding X. Liu

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

III - Viral Assembly and Release | Pp. 199-202

Efficient Transduction of Dendritic Cells Using Coronavirus-Based Vectors

Klara K. Eriksson; Divine Makia; Reinhard Maier; Luisa Cervantes; Burkhard Ludewig; Volker Thiel

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

III - Viral Assembly and Release | Pp. 203-206

Insights from the Association of SARS-CoV S-Protein with its Receptor, ACE2

Wenhui Li; Hyeryun Choe; Michael Farzan

In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.

IV - Viral Entry | Pp. 209-218

Attachment Factor and Receptor Engagement of Sars Coronavirus and Human Coronavirus NL63

Heike Hofmann; Andrea Marzi; Thomas Gramberg; Martina Geier; Krzysztof Pyrc; Lia van der Hoek; Ben Berkhout; Stefan Pöhlmann

In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.

IV - Viral Entry | Pp. 219-227

Interactions Between Sars Coronavirus and its Receptor

Fang Li; Wenhui Li; Michael Farzan; Stephen C. Harrison

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 229-234

Proteolysis of Sars-Associated Coronavirus Spike Glycoprotein

Graham Simmons; Andrew J. Rennekamp; Paul Bates

So far, we have only considered functions on the real line. We have seen how to hide those annoying єs and δs in the definition of continuity, replacing them with open sets. This enables us to consider functions with domains and ranges different from R; all we need is some notion of “open set”.

IV - Viral Entry | Pp. 235-240

Fluorescence Dequenching Assays of Coronavirus Fusion

Victor C. Chu; Lisa J. McElroy; Beverley E. Bauman; Gary R. Whittaker

In the natural world, individual organisms can adapt as their environment changes. In most evolution, however, individual organisms tend to consist of rigid solutions, with all adaptation occurring at the population level. If we are to use artificial evolving systems as a tool in understanding biology or in engineering robust and intelligent systems, however, they should be able to generate solutions with fitness-enhancing phenotypic plasticity. Here we use Avida, an established digital evolution system, to investigate the selective pressures that produce phenotypic plasticity. We witness two different types of fitness-enhancing plasticity evolve: plasticity, in which the same sequence of actions produces different results depending on the environment, and plasticity, where organisms choose their actions based on their environment. We demonstrate that the type of plasticity that evolves depends on the environmental challenge the population faces. Finally, we compare our results to similar ones found in vastly different systems, which suggest that this phenomenon is a general feature of evolution.

IV - Viral Entry | Pp. 241-246