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Palabras clave – provistas por la editorial

pluripotent stem cells; cardiomyocytes; cardiac tissue engineering; human heart; tissue maturation; Wilms’ tumor suppressor 1; cardiomyocyte differentiation; mouse embryonic stem cells; myocardial infarction; YAP; TEAD1; Toll-like receptor; heart; TLR4; cardiomyocyte; innate immune responses; Wilms’ tumor suppressor 1 (Wt1); cardiac development; coronary vessel formation; transcriptional regulation; cardiac malformation; epicardium; epicardial derived cells (EPDCs); epithelial mesenchymal transition (EMT); cardiac cell fate; regeneration; arrhythmogenic cardiomyopathy (ACM); lamin A/C; atomic force microscopy (AFM); cell–cell adhesion; neonatal rat ventricular fibroblasts (NRVF); tunneling nanotubes (TNT); gene therapy; cardiovascular diseases; cardiac regeneration; cardiac reprogramming; therapeutic angiogenesis; growth factors; reactive oxygen species; apoptosis; Bmp signaling; Hand1; Smad; heart development; SH3BGR; cardiac hypertrophy; SRF signaling; Hippo signaling; cardiomyogenic differentiation; cardiac mesenchymal stromal cell; hydrogels; histone deacetylase inhibitors; translational studies; gene expression and regulation; cardiomyopathy; heart failure; reverse remodelling; left ventricular assist device; n/a