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Palabras clave – provistas por la editorial

rheumatoid arthritis; exercise therapy; autoimmune disorder; treadmill running; exercise; articular cartilage; collagen-induced arthritis; pro-inflammatory cytokine; connexin 43; osteoporosis; microenvironment; inflammation; mesenchymal stem/stromal cells; bone injury; repair/regeneration; microparticles; joint inflammatory diseases; Wnt signaling; Dkk-1; biologics; ankylosing spondylitis; axial spondyloarthritis; bone homeostasis; osteoarthritis; aging; PTOA; gene expression; RNA-seq; cartilage degeneration; scRNA-seq; chondrocytes; animal model; pain; melatonin; angiotensin II; arthritis; bone erosion; tumor necrosis factor; renin-angiotensin system; angiotensin II type 1 receptor; psoriatic arthritis; osteoclastogenesis; miR-941; WNT16; polymorphism; genetic predisposition; mutation; methotrexate; interleukin; targeted therapy; interstitial lung disease; subclinical involvement; ACPA; pulmonary function testing; CPET; surfactant protein D; DLCO; HRCT; lymph node stromal cells; tolerance; autoimmunity; citrullination; macrophages; PADI2; inflammatory cytokines; adhesion; apoptosis; L5; macrophage foam cell; CD11c expression; atherosclerosis; rheumatoid arthritis (RA); synovium; cytokines; HHV-7; PCR; ELISA; immunohistochemistry; adipocyte; infrapatellar fat pad; anterior cruciate ligament; cartilage; whole transcriptome sequencing; biomarker; CRISPR/Cas9; miRNA; genome editing; autoimmune arthritis; Nkx2-3; B cell activation; Spondyloarthritis; ankylosing arthritis; interleukin-17; interleukin-23; tibial compression; gut microbiome; antibiotics; LPS; adults; juvenile; immune; laser blood irradiation; low-level laser; systemic; naïve rheumatoid arthritis; CD4+ T-lymphocytes; methotrexate response; IFNγ, IL-17A; STAT expression; osteonecrosis; taurine; mitochondrial function; glucocorticoid; discoidin domain receptors 1 (Ddr1); osteoarthritis (OA); autophagy; terminal differentiation; Treg; Th17; microRNA; transcriptional factor; JAK/STAT; Janus kinase inhibitors; cardiovascular system; heart failure; thromboembolic; lipid profile disturbances; treatment; molecular mechanisms; biomarkers; prevention; collagen triple helix repeat containing 1; CTHRC1; cartilage destruction; fibroblast-like synoviocytes