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Palabras clave – provistas por la editorial

glioblastoma multiforme; proton therapy; combined treatments; gene signatures; brain arteriovenous malformation; endothelial cells; ionizing radiation; proteomics; stereotactic radiosurgery; vascular targeting; DNA repair; hypoxia; modeling; radiosensitizer; Monte Carlo simulation; Geant4/Geant4-DNA; X-rays; microdosimetry; nanodosimetry; DSB yield; radiation track chemistry; chemical track structure; oxygen effect; oxygen depletion; ion beam therapy; ROS; superoxide anion; Monte Carlo radiation transport; complex DNA damage coupled with base damage; modelling of DNA damage yields; amino acid; valine; MeV ion irradiation; radiolysis; infrared absorption spectroscopy; destruction cross section; stopping power dependence; carbon ions; radiation-induced damage; chromatin structure; electron microscopy; CLEM; DNA and RNA cytochemistry; Osmium ammine B; ChromEMT; DNA-specific staining; high-dose ionizing radiation; RNA-Seq; differential gene expression; DNA damage response; triple-negative breast cancer; CHK1; Rad51; direct and indirect damage; DMSO; radicals; radical scavengers; Glioblastoma; DNA damage; head-and-neck tumors; ion beam radiotherapy; relative biological effectiveness (RBE); RBE models; protons; Monte Carlo simulations; embryo; gastrulation; preimplantation; apoptosis; glioblastoma; stem cells; metabolism; MRS; photon beams; pencil beam scanning; double scattering; cell survival; radiation doses; repair mechanism; translocation; transition transversion ratio; IR-induced variants; SNVs; InDels; topological associating domains; melatonin; oxidative stress; radioprotection; reactive oxygen species; vitamin D; radiation biology; radiation protection; health effects; epigenetics; low dose radiation; DNA methylation; non-targeted effects; triple-negative breast cancer (TNBC); xenograft mice; microarray; environmental radiation; hibernation; HIF-1; hypothermia; radioresistance; space travel; FLASH; proton beam therapy; radiotherapy; radiobiology; n/a