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Redox Active Molecules in Cancer Treatments
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Palabras clave – provistas por la editorial
oxygen detection; dendrogram analysis; hierarchical clustering; cancer cells; luminescence lifetime; hydrogen peroxide; oxidative stress; photodynamic therapy; time-resolved imaging; breast cancer; NRF2; polyphenols; melatonin; acute myeloid leukemia (AML); metformin; MCL-1 inhibitor S63845; reactive oxygen species (ROS); AQP3; AQP5; Anneslea fragrans; antioxidant; guided isolation; flavonoid glycosides; spheroids; HT29 cells; cluster analysis; principal component analysis; hydrolysable tannins; HHDP-digalloylglucose isomer; dihydroxyphenyl-γ-valerolactone; autophagy; colorectal cancer cells; reactive oxygen species; endoplasmic reticulum stress; pentoxifylline; astaxanthin; particle encapsulation; ionizing radiations; macrophages; radiation-induced fibrosis; Rhopilema nomadica; apoptosis; cell cycle arrest; HepG2; soybean; peptides; antioxidant capacity; magnetite nanoparticles; conjugation; multicomponent reaction; copper complexes; glutathione depletion; cysteine; Casiopeina; intermediate; copper reduction; papaverine; cancer; morphology; proliferation; cell cycle; chain-transfer agent; chemotherapy; free radical chain reaction; lipid peroxidation; lipophilic thiol; oxidative cell death; prooxidative drug; radical propagation; rate-limiting step; mitochondria; bioenergetics; anticancer; cheminformatics; in silico; Keap1; molecular docking; molecular dynamics; myeloperoxidase; NADPH oxidase; seed antioxidant peptide; xanthine oxidase; osteosarcoma; naringenin; ROS; ER stress; ferroptosis; inducers; inhibitors; drug-likeness; neurodegenerative; polyphenol; n/a
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libros
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Suiza