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Genetics and Epigenetics in Complex Diseases

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Palabras clave – provistas por la editorial

chromatin remodeling; circular RNA; DNA methylation; histone modification; non-coding RNA; oncogenes; tumor suppressor genes; tumor virus; chronic lymphocytic leukemia; CLL; SF3B1 mutation; NOTCH; IKAROS; syncope; vasovagal syncope; genetics; complex disorders; susceptibility; twin studies; family studies; candidate gene association studies; genome-wide studies; acute myeloid leukemia; pediatric; trio sequencing; germline cancer predisposition; POT1; shelterin complex; psoriasis; psoriatic arthritis; NF-κB; comorbidities; genetic architecture; SKP1; CUL1; RBX1; SKP2; FBXW7; FBXO5; SCF complex; genome instability; chromosome instability; cancer; VDBP; vitamin D binding protein; rs7041; rs4588; bone density; diabetes; obesity; COPD; pulmonary tuberculosis; SNP; MD; PD; CD33; Alzheimer’s disease; sialic acid; phagocytosis; KLF14; body shape indices; metabolic traits; differential effect; genetic variants; resistin; soluble suppression of tumorigenicity 2; weighted genetic risk score; Taiwan Biobank; coronary artery disease; all-cause mortality; major adverse cardiac events; acute lymphoblastic leukemia; RAD21; cohesin complex; n/a

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Suiza