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Palabras clave – provistas por la editorial

palbociclib; estrogen receptor; breast cancer; CDK4/6; CDK4/6 inhibitors; therapy resistance; DNA repair; metabolic rewiring; quantitative proteomics; urothelial carcinoma; molecular subtypes; therapeutic target; prostate cancer; prognostic signature; diagnostic signature; biomarkers; cancer progression; aggressive cancer; androgen receptor; gene expression; insulin receptor; germline mutations; somatic mutations; PTEN; TMPRSS2; ERG; androgen receptors; cervical cancer; alternative splicing; SR proteins; hnRNP; drug resistance; head and neck squamous cell carcinoma (HNSCC); aberrant splicing events; human papillomavirus (HPV) infection; non-coding RNA (ncRNA); methylation; mutational burden; next-generation digital histopathology; tissue cytometry; multiplexing; RNA ISH; cancer; tumor immune microenvironment; tumor microenvironment; IRX4; transcript; isoforms; long non-coding RNA; single nucleotide polymorphisms; genome-wide association studies