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Palabras clave – provistas por la editorial

Candida albicans; acridine; antifungal; topoisomerase; inhibitor; candidiasis; 1,3,4-oxadiazole; drug discovery; antifungal agents; drug resistance; toxicity; biofilm; invasive Candida infections; invasive fungal infections; antifungal prophylaxis; newborns; surgery; neonatal surgery; multivalency; anti-adhesion glycoconjugates; glycomimetics; Candida; C. albicans; XCL1; metamorphic protein; fold-switching protein; antifungal peptide; polymorphism; hyphal morphogenesis; hyphal activation; signal transduction pathways; cell cycle regulation; sphingolipids; myriocin; fungal infections; epigenetic; yeast; chromatin; hyphae; human fungal pathogen; nutrient sensing; amino acid metabolism; proline catabolism; mitochondria; SPS-sensor; nitrogen catabolite repression; glucose repression; immunometabolism; metabolism; macrophages; epithelial cells; glycolysis; glucose; moonlighting proteins; n/a