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Palabras clave – provistas por la editorial

COVID-19; fibrinolysis; renin-aldosterone-angiotensin-system (RAAS); plasminogen activator inhibitor 1 (PAI-1); urokinase plasminogen activator; urokinase plasminogen activator receptor; plasminogen activator inhibitor-1; acute lung injury and repair and pleural injury and pleural organization; PAI-1; cardiovascular disease; cancer; inflammation; fibrosis; aging; obstructive sleep apnoea; OSA; coagulation; platelets; brown adipose tissue; obesity; plasminogen activation; immune response; thrombin activatable fibrinolysis inhibitor; TAFI; proCPU; proCPB; proCPR; carboxypeptidase; uPA; uPAR; PA system; tissue-type plasminogen activator (tPA); urokinase-type plasminogen activator (uPA); neurodegeneration; thrombin; shear; clot retraction; Factor XIII; clot stability; NETs; plasmin; plasminogen activator; PAI-2; antiplasmin