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Palabras clave – provistas por la editorial

Alzheimer disease; expression quantitative trait loci (eQTL); rare variants; set-based eQTL; SKAT-O; pathways; immune system; inflammation; ROSMAP; ADNI; complement; complement receptor 1; clusterin; late-onset Alzheimer’s disease; genetics; neuroinflammation; Alzheimer’s disease; cytokines and growth factors; genome-wide association study (GWAS); Polygenic Risk Score (PRS)-based analysis; TOMM40 gene; RNA transcription; pseudogene; mitochondrial dysfunction; ITIM; ITAM; SIGLEC14; SIGLEC5; copy number variation; CNV; GWAS; long non-coding RNA; biomarkers; peripheral system; amyloid β; tau; APOE; TREM2; OMICS; therapeutics; pairwise feature correlation; feature reduction; machine learning; n/a