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Palabras clave – provistas por la editorial

spumavirus; feline illness; proviral load; neglected virus; bovine foamy virus; infectious clone; particle release; cell-free transmission; foamy virus; spumaretrovirus; cross-species virus transmission; zoonosis; restriction factors; immune responses; FV vectors; virus replication; latent infection; feline foamy virus; epidemiology; retrovirus; Spumaretrovirus; mountain lion; Puma concolor; ELISA; protease; reverse transcriptase; RNase H; reverse transcription; antiviral drugs; resistance; simian foamy virus; gibbon; lesser apes; co-evolution; complete viral genome; equine foamy virus; isolation; Japan; sero-epidemiology; reptile foamy virus; endogenous foamy virus; endogenous retrovirus; ancient retroviruses; co-speciation; foamy virus-host interactions; viral tropism; infection; kidney; cats; chronic kidney disease; chronic renal disease; integrase; integration; co-infections; NHP; pathogenesis; zoonoses; viral prevalence; Neotropical primates; free-living primates; Brazil; new world primates; simian retrovirus; BFV; spuma virus; model system; animal model; animal experiment; miRNA function; gene expression; antiviral host restriction; gene therapy; in-vivo gene therapy; hematopoietic stem and progenitor cells; foamy virus vector; pre-clinical canine model; SCID-X1; innate sensing; cGAS; STING; foamy viruses; wild ruminants; European bison; red deer; roe deer; fallow deer; seroreactivity; inter-species transmission; HSC; gene marking; FV gene transfer to HSCs; gene therapy alternatives; serotype; high-throughput sequencing; replication kinetics; cytopathic effect; reverse transcriptase activity; miRNA expression; virus-host-interaction; miRNA target gene identification; innate immunity; ANKRD17; Bif1 (SH3GLB1); replication in vitro