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Palabras clave – provistas por la editorial

CTCL; apoptosis; cell viability; c-FLIP; XIAP; artemisinin; SH-SY5Y cells; hippocampal neurons; H2O2; AMPK pathway; atherosclerosis; sphingomyelin synthase 2; endothelial dysfunction; endoplasmic reticulum stress; β-catenin; insulin resistance; cancer; cardiovascular disease; neurodegenerative disorders; exercise; mitochondria; oxidative stress; PGC-1; Nrf2; UCPs; ROS; light; DNA damage; evolution; D-box; cavefish; Spalax; trimethylamine N-oxide; cardiomyocytes; cardiotoxicity; mitochondrial membrane potential; CORM-2; NADPH oxidase; AP-1; HO-1; Renal cell carcinoma (RCC); reactive oxygen species (ROS); glutathione (GSH) metabolism; cancer therapy; clear cell RCC; papillary RCC; chromophobe RCC; sarcopenia; reactive oxygen species; redox signaling; antioxidant supplementation; protein aggregation; redox; proteinopathy; peroxiredoxins; tumorigenesis; ROS scavengers; n/a