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Palabras clave – provistas por la editorial

soy; isoflavones; mammary tumor prevention; rodent models; chemical carcinogens; transgenic mice; Zebrafish; Drosophila; rats; mice; NPM-1; FLT3 ITD; ETO-1; IDH1/2; neural stem cells; brain and nervous system cancers; neurogenic niches; radiotherapy; sparing of neurogenic regions; carcinoma; consensus molecular subtypes; intestine; oncogenes; signaling cascades; tumor suppressors; tumorigenesis; MPN (myeloproliferative neoplasms); zebrafish; iPSCs; JAK2; MPL; CALR; thrombosis; ubiquitin–proteasome system; cancer; mouse model; gene inactivation; colorectal cancer; mouse models; microbiota; antitumor immunity; melanoma; mutation; genetics; animal model; swine; MeLiM; progression; spontaneous regression; devitalization; metaplasia; Cdx; animal models; epigenetics; xenotransplantation; drug screen; pre-clinical cancer model; non-mouse models; gene editing; stem cells; solid tumors; hematologic malignancies