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Palabras clave – provistas por la editorial

inhibitor; metabolic stability; α-methylation; oxoesters; phospholipase A2; adrenic acid; arachidonic acid; mass spectrometry; lipid signaling; inflammation; monocytes/macrophages; crotoxin; snake venom; lung impairment; inflammatory response; lipid mediators; neuromuscular blocker; lipidomics; PAP-2; autotaxin; lysophosphatidate; G protein-coupled receptor; PLA2G6; fatty liver; phospholipid remodeling; diet-induced obesity; morbidly obesity; choline and methionine deficiency; glioblastoma; sphingolipid; sphingosine-1-phosphate; sphingomyelinase; sphingomyelin; metastasis; phosphatidic acid; diacylglycerol; lipin; signaling; cPLA2α; psoriasis; proliferation; anti-inflammatory; atherosclerosis; phospholipases; macrophages; T cells; lipins; pancreatic islets; β-cells; insulin secretion; glucose tolerance; insulin resistance; group VIA phospholipase A2; fatty acid; knockout mouse; lipid mediator; lysophospholipid; membrane; phospholipid; ceramide; acidic sphingomyelinase; neutral sphingomyelinase; hepatocellular carcinoma; alcoholic and nonalcoholic steatohepatitis; preadipocytes; prostaglandins; adipokines; cytokines; EP receptors; Group V phospholipase A2; lipids; majeed syndrome; LPIN2; LIPIN2; chronic non-bacterial osteomyelitis; chronic recurrent multifocal osteomyelitis; autoinflammatory; inflammasome; macrophage; osteoclast; n/a