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Palabras clave – provistas por la editorial

diabetic kidney disease; caloric restriction; NADH/NAD+; redox imbalance; mitochondrial homeostasis; mitophagy; oxidative stress; kidney allograft; kidney rejection; ischemia; acute kidney injury (AKI); chronic kidney disease (CKD); tricarboxylic acid (TCA) cycle; mitochondrial metabolism; mitochondrial redox signaling; mitochondrial proteins; oxidative phosphorylation (OXPHOS); fatty acid (FA) β-oxidation; mitochondrial dynamics; biogenesis; diabetes; kidney; mitochondria; Oryza sativa; rice husk; TCA cycle metabolites; kidney diseases; renalase; chronic kidney disease; major adverse cardiovascular outcomes; cadmium; kidney injury; renal toxicity; oxidative damage; proximal tubule; controlled oxygenated rewarming; mitochondrial uncoupling; rewarming injury; temperature paradox; redox; mitochondrial dysfunction; SGLT2; mitochondrial reactive oxygen species; Warburg effect; podocytopathies; mitochondrial oxidative stress; reactive oxygen species (ROS); antioxidant defense; cell death; n/a