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Palabras clave – provistas por la editorial

selenoproteome; selenoprotein hierarchy; nonradioactive isotopes; SEC-ICP MS; glutathione peroxidase; thioredoxin reductase; SECIS; translation regulation; autoimmune thyroid disease; diabetes mellitus; Graves’ disease; Hashimoto thyroiditis; infection; inflammation; long-COVID; rheumatoid arthritis; selenoprotein P; sepsis; selenoprotein W; thioredoxin; 14-3-3; Akt; cell death; Selenof; selenium; selenoprotein; colon cancer; barrier integrity; cardiovascular; heart; selenoproteins; Keshan’s Disease; bacteria; selenite; selenium delivery system; Trsp; hypothalamus; Agrp neuron; sex differences; diet-induced obesity; leptin resistance; macrophage; differentiation; redox signaling; NRF2; NF-κB; lipid mediators; seleocysteine; autoimmunity; lymphocyte; cadmium cytotoxicity; cancer therapy; cisplatin; ICP-MS; nonsynonymous mutation; selenium homeostasis; ZIP8; Trit1; isopentenylation; tRNA[Ser]Sec; selenocysteine; genetic variance; human disease; selenophosphate synthetase; endothelial cell; reactive oxygen species; cell growth; angiogenesis; SEPHS1; early embryogenesis; embryonic lethality; prostate; cancer; tumor suppressor; selenoprotein deficiency; SECISBP2; Sec-tRNA[Ser]Sec; SEPSECS; antioxidative defense; autoantibody; Hashimoto’s thyroiditis; trace element; recoding; SECIS-binding protein; translation termination; nonsense-mediated decay; ribosome rescue; health; mouse models; selenocysteine (Sec); virus; viral; antioxidant; HIV; HCV; HBV; coxsackie virus; influenza; n/a; HIV-1; viral infection; SELENOS; SELENOO; primary T cells; Jurkat; SupT1; translational control