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Palabras clave – provistas por la editorial

receptor activator of nuclear factor ?B; infliximab; tripterine; triptolide; osteoblast; tumor necrosis factor-alpha; synovial cell; anti-arthritis; biosimilars; Epstein-Barr virus; cytokines; SOX9; parathyroid hormone; nitric oxide; rat; etanercept; angiogenesis; glycosylation; mitogen activated protein kinase; Th9 lymphocytes; rheumatoid arthritis; IL-6; clodronate; bone erosion; mesenchymal stem cells; collagen-induced arthritis; biological; gene expression; inflammatory arthritis; osteoarthritis; fraxinellone; nuclear factor kappa B; messenger RNA; inflammation; miRNA; disease-modifying; adipokines; WNT; glycoprotein 42; miR-199a-5p; proliferation; next-generation sequencing; collagen; osteoarthritis (OA); experimental arthritis; bone morphogenetic protein; TNF-?; computational modeling; basic research; osteoclast; therapeutics; certolizumab pegol; chondrocytes; progenitor cells; adjuvant arthritis; adalimumab; triterpenoid; sclareol; TNF?; fibroblast growth factor 2; antibodies; osteoblasts; molecular pathology; Th17; immunology; obesity; visfatin; articular cartilage; autoimmune; biomarkers; celastrol; MAPK; disease pathways; IL1?; arthritis; bioinformatics; anticitrullinated peptide antibodies; drug delivery system; antagonists; shared epitope; pathology; SMA- and MAD-related protein; small-molecule inhibitor; transforming growth factor ?; mice; golimumab; spinal fusion; antirheumatic drug; early osteoarthritis; stem cell; rheumatoid factor; therapeutic antibody; bisphosphonate; osteoclastogenesis; interleukin; spondyloarthropathies; clinical translation; therapy; Traditional Chinese medicine; chemokines; structure; cell signaling; microRNA