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Nature
Resumen/Descripción – provisto por la editorial en inglés
Nature is a weekly international journal publishing the finest peer-reviewed research in all fields of science and technology on the basis of its originality, importance, interdisciplinary interest, timeliness, accessibility, elegance and surprising conclusions. Nature also provides rapid, authoritative, insightful and arresting news and interpretation of topical and coming trends affecting science, scientists and the wider public.Palabras clave – provistas por la editorial
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Disponibilidad
Institución detectada | Período | Navegá | Descargá | Solicitá |
---|---|---|---|---|
No detectada | desde jul. 2012 / hasta dic. 2023 | Nature.com | ||
No detectada | desde jul. 2006 / hasta ago. 2012 | Ovid |
Información
Tipo de recurso:
revistas
ISSN impreso
0028-0836
ISSN electrónico
1476-4687
Editor responsable
Springer Nature
País de edición
Reino Unido
Fecha de publicación
1869-
Tabla de contenidos
Endothelial sensing of AHR ligands regulates intestinal homeostasis
Benjamin G. Wiggins; Yi-Fang Wang; Alice Burke; Nil Grunberg; Julia M. Vlachaki Walker; Marian Dore; Catherine Chahrour; Betheney R. Pennycook; Julia Sanchez-Garrido; Santiago Vernia; Alexis R. Barr; Gad Frankel; Graeme M. Birdsey; Anna M. Randi; Chris Schiering
Palabras clave: Multidisciplinary.
Pp. No disponible
How to be successful as a research mathematician? Follow your gut
Davide Castelvecchi
Palabras clave: Multidisciplinary.
Pp. 485-486
Evelyn Boyd Granville, space-flight trailblazer (1924—2023)
Mar Hicks
Palabras clave: Multidisciplinary.
Pp. 487-487
How to reduce Africa’s undue exposure to climate risks
Asaf Tzachor; Catherine E. Richards; Masilin Gudoshava; Patricia Nying’uro; Herbert Misiani; Jemimah G. Ongoma; Yoav Yair; Yacob Mulugetta; Amadou T. Gaye
Palabras clave: Multidisciplinary.
Pp. 488-491
Can oyster farming help save the planet?
Linda Nordling
Palabras clave: Multidisciplinary.
Pp. 690-690
COVID infection risk rises the longer you are exposed — even for vaccinated people
Anil Oza
Palabras clave: Multidisciplinary.
Pp. No disponible
A pentameric TRPV3 channel with a dilated pore
Shifra Lansky; John Michael Betancourt; Jingying Zhang; Yining Jiang; Elizabeth D. Kim; Navid Paknejad; Crina M. Nimigean; Peng Yuan; Simon Scheuring
Palabras clave: Multidisciplinary.
Pp. No disponible
Widespread retreat of coastal habitat is likely at warming levels above 1.5 °C
Neil Saintilan; Benjamin Horton; Torbjörn E. Törnqvist; Erica L. Ashe; Nicole S. Khan; Mark Schuerch; Chris Perry; Robert E. Kopp; Gregory G. Garner; Nicholas Murray; Kerrylee Rogers; Simon Albert; Jeffrey Kelleway; Timothy A. Shaw; Colin D. Woodroffe; Catherine E. Lovelock; Madeline M. Goddard; Lindsay B. Hutley; Katya Kovalenko; Laura Feher; Glenn Guntenspergen
<jats:title>Abstract</jats:title><jats:p>Several coastal ecosystems—most notably mangroves and tidal marshes—exhibit biogenic feedbacks that are facilitating adjustment to relative sea-level rise (RSLR), including the sequestration of carbon and the trapping of mineral sediment<jats:sup>1</jats:sup>. The stability of reef-top habitats under RSLR is similarly linked to reef-derived sediment accumulation and the vertical accretion of protective coral reefs<jats:sup>2</jats:sup>. The persistence of these ecosystems under high rates of RSLR is contested<jats:sup>3</jats:sup>. Here we show that the probability of vertical adjustment to RSLR inferred from palaeo-stratigraphic observations aligns with contemporary in situ survey measurements. A deficit between tidal marsh and mangrove adjustment and RSLR is likely at 4 mm yr<jats:sup>−1</jats:sup> and highly likely at 7 mm yr<jats:sup>−1</jats:sup> of RSLR. As rates of RSLR exceed 7 mm yr<jats:sup>−1</jats:sup>, the probability that reef islands destabilize through increased shoreline erosion and wave over-topping increases. Increased global warming from 1.5 °C to 2.0 °C would double the area of mapped tidal marsh exposed to 4 mm yr<jats:sup>−1</jats:sup> of RSLR by between 2080 and 2100. With 3 °C of warming, nearly all the world’s mangrove forests and coral reef islands and almost 40% of mapped tidal marshes are estimated to be exposed to RSLR of at least 7 mm yr<jats:sup>−1</jats:sup>. Meeting the Paris agreement targets would minimize disruption to coastal ecosystems.</jats:p>
Palabras clave: Multidisciplinary.
Pp. No disponible
Gut microbial carbohydrate metabolism contributes to insulin resistance
Tadashi Takeuchi; Tetsuya Kubota; Yumiko Nakanishi; Hiroshi Tsugawa; Wataru Suda; Andrew Tae-Jun Kwon; Junshi Yazaki; Kazutaka Ikeda; Shino Nemoto; Yoshiki Mochizuki; Toshimori Kitami; Katsuyuki Yugi; Yoshiko Mizuno; Nobutake Yamamichi; Tsutomu Yamazaki; Iseki Takamoto; Naoto Kubota; Takashi Kadowaki; Erik Arner; Piero Carninci; Osamu Ohara; Makoto Arita; Masahira Hattori; Shigeo Koyasu; Hiroshi Ohno
<jats:title>Abstract</jats:title><jats:p>Insulin resistance is the primary pathophysiology underlying metabolic syndrome and type 2 diabetes<jats:sup>1,2</jats:sup>. Previous metagenomic studies have described the characteristics of gut microbiota and their roles in metabolizing major nutrients in insulin resistance<jats:sup>3–9</jats:sup>. In particular, carbohydrate metabolism of commensals has been proposed to contribute up to 10% of the host’s overall energy extraction<jats:sup>10</jats:sup>, thereby playing a role in the pathogenesis of obesity and prediabetes<jats:sup>3,4,6</jats:sup>. Nevertheless, the underlying mechanism remains unclear. Here we investigate this relationship using a comprehensive multi-omics strategy in humans. We combine unbiased faecal metabolomics with metagenomics, host metabolomics and transcriptomics data to profile the involvement of the microbiome in insulin resistance. These data reveal that faecal carbohydrates, particularly host-accessible monosaccharides, are increased in individuals with insulin resistance and are associated with microbial carbohydrate metabolisms and host inflammatory cytokines. We identify gut bacteria associated with insulin resistance and insulin sensitivity that show a distinct pattern of carbohydrate metabolism, and demonstrate that insulin-sensitivity-associated bacteria ameliorate host phenotypes of insulin resistance in a mouse model. Our study, which provides a comprehensive view of the host–microorganism relationships in insulin resistance, reveals the impact of carbohydrate metabolism by microbiota, suggesting a potential therapeutic target for ameliorating insulin resistance.</jats:p>
Palabras clave: Multidisciplinary.
Pp. No disponible