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The Role of Toll-Like Receptors (TLR) in Infection and Inflammation

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Palabras clave – provistas por la editorial

hepatitis C virus; infection; innate immunity; Toll-like receptor; cytokines; Omicron; spike protein; SARS-CoV-2; COVID-19; cytokine storm; NF-kappaB; Toll-like receptor (TLR); monoclonal antibody TLR4; compression force; MAPKs; AKT; human PDL; sterile inflammation; TLR; immune system; inflammation; antiviral; polymorphism; sepsis; septic shock; TLR7; TLR8; 2′-O-ribose-methylation; RNase T2; immune activation; CD14; LPS; hop; TLR4; oligonucleotide; sncRNA; endocytosis; broad-spectrum; antiviral agent; nucleolin; virus entry; immunoregulation; RNA therapeutics; TLR7 (Toll-like receptor 7); MUC1 (Mucin 1); aluminum adjuvant; tumor vaccine; immunotherapy; toll-like receptor-2 (TLR2); advanced glycation end products (AGEs); aquaporin-3 (AQP3); histone deacetylase inhibitor; diabetes; keratinocytes; skin; n/a; TLR4–RAGE crosstalk; glucose; lipopolysaccharide (LPS); inflammatory; alveolar macrophages

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libros

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Suiza