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Palabras clave – provistas por la editorial

steroidogenesis; testis; transcription; testosterone; Leydig cell; GATA; cholesterol; cholesterol transport; steroidogenic acute regulatory protein; sterol carrier protein 2; sterol carrier protein-x; MnHR4; 20-hydroxyecdysone (20E); Macrobrachium nipponense; molt; RNA interference; Leydig cells; star; AMPK; phosphoproteomics; proteomics; activator protein 1; CREB; CEBPB; Steroidogenesis; signaling cascade; Nuclear receptors; bZIP factors; MEF2 factors; GATA4; CAMKI; PKA; ERK1/2; Hippo signaling; Lats1/2; fetal Leydig cells; Sertoli cells; transgenic mouse model; ovary; cow; Hippo; follicle deviation or divergence; yes-associated protein 1; CTGF; CYR61; ANKRD1; transgenic mice; adrenal cortex; development; maintenance; glucocorticoid receptor; androgens; leydig cells; AAV9; insulin-like 3; transcription factors; linker-scanning mutagenesis; DNA–protein interactions; bile acids; TGR5; cholesterol esters; LH; LH-receptor; spermatogenesis; thrombospondin-2; NR5A1; enhancer; pituitary gonadotrope; luteinizing hormone; follicle-stimulating hormone; 11-oxygenated androgens; steroid hormones; LC-MS/MS; method validation; adrenocortical carcinoma; histone deacetylase inhibitor; cell differentiation; gene expression; adrenal gland inner cortex; X-zone; DHCR24; seladin-1; endocrine disruptors; EDC mixtures; in utero exposures; transcriptome analysis; testicular function; aldo-keto reductase; bile acid; deficiency; congenital adrenal hyperplasia; hydroxysteroid dehydrogenase; prostate cancer; single nucleotide polymorphism; structure-function; steroid reductase; pseudohermaphroditism