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Palabras clave – provistas por la editorial

insulin secretory granule; beta-cells; granule protein purification; free fatty acid receptor (FFA) 2; FFA3; gut microbiome; incretin; insulin secretion; short-chain fatty acids; type 1 diabetes; cytosolic calcium concentration; glucose; metabolic amplification; mitochondria; nutrient secretagogues; diabetes; hypoglycemic unaware; ischaemia; islet cell transplantation; organ donation; pancreas; insulin; beta cell; human; islet; polarisation; machine learning; deep learning; cell segmentation; automation; pancreatic islets; leptin; exocytosis; tissue-engineering; in vivo imaging; metabolism; β-cell; viral transduction; transplantation; amplifying pathway; hyperglycemia; adenylyl cyclase; incretins; glucokinase; forskolin; cAMP; exenatide; islets; bioenergetics; glucose-stimulated insulin secretion; respiration; advanced glycation end products; alagebrium chloride; cross-link breaker; immunopeptidome; MIN6N8 cell line; NOD mouse; autoimmune diabetes; pancreatic beta-cells; intrauterine growth restriction; maternal obesity; developmental programming; islet amyloid polypeptide (IAPP); granin; secretory pathway; trans-Golgi network (TGN); granule; pancreatic β-cell; amino acid; aminoaciduria; diabetes incidence; insulitis; neutral amino acid transporter; non-obese diabetic mouse; pancreatic islet; Slc6a19 deficiency; n/a