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Palabras clave – provistas por la editorial

neutrophils; lymphocytes; NLR; multiple sclerosis; disease activity; inside-out; outside-in; oligodendrocytosis; demyelination; gliosis; histology; top-down proteomics; bioinformatics; mitochondria; CD4+ T cells; memory T cells; autoimmune disease; effector memory T cell; central memory T cell; tissue-resident T cell; experimental autoimmune encephalomyelitis; monocytes; granulocyte-macrophage colony-stimulating factor; S100B; relapsing–remitting experimental autoimmune encephalomyelitis; pentamidine; NG2-glia; progenitors; lineage; in utero electroporation; morphometric analyses; clonal analyses; lesioned brain; sphingosine-1-phosphate receptors; glutamate synaptic dysfunction; microglia; T lymphocytes; experimental autoimmune encephalomyelitis (EAE); pro-inflammatory cytokines; neuroinflammation; ozanimod; AUY954; A971432; S1P1; S1P5; kynurenine pathway; kynurenic acid; oxidative stress; quinolinic acid; N-acetylserotonin; IDO; NAD+, multiple sclerosis; laquinimod; n/a