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Palabras clave – provistas por la editorial

endothelial cells; oxidative stress; inflammageing; endothelial dysfunction; aldehyde dehydrogenase-2; cardiovascular disease; neurovascular disease; vascular inflammation; APE1/Ref-1; cardiovascular diseases; subcellular localization; serological biomarkers; atherosclerosis; aerobic exercise; PCSK9; LOX-1; insulin resistance; macrophage polarity; serum γ-glutamyltransferase; essential hypertension; cardiovascular risk factors; angiogenesis; nargenicin A1; compound 9; VEGF; VEGFR2; HIF-1α; electronegative low-density lipoprotein; LDL(–); L5 LDL; oxidized LDL; oxLDL; ADMA; arginine; arginine paradox; BH4; blood pressure; COVID-19; dietary supplements; endothelium; eNOS uncoupling; heart failure; hypertension; L-arginine; myocardial infarction; NADPH; nitric oxide; peripheral artery disease; ApoE knockout mouse; atorvastatin; VCAM-1; galectin-3; neutrophil/lymphocyte ratio; electronegative LDL; LDL(−); lectin-like oxLDL receptor-1; dyslipidemia; chronic venous insufficiency; prolyl oligopeptidase (POP); inflammation; endothelial disfunction; lung disease; endothelial to mesenchymal transition; pulmonary hypertension; pulmonary fibrosis; metformin; diabetes; CV risk; hyperglycemia; glycated lipoproteins; glycated LDL; glycated HDL; endothelial cell dysfunction; molecular mechanisms; epigenetic factors; therapeutic approaches; vasoactive factors; EndoMT; TGF-β; epigenetics; endothelial cell; glycolysis; metabolism; pathological angiogenesis; tumor microenvironment; MRI (magnetic resonance imaging); high-fat diets; plaque burden; low-level laser therapy; phototherapy; vascular disease; healing; ischemia; vascular calcification; chronic kidney disease; CKD; uremic toxins; hyperphosphatemia; vascular smooth muscle cells; VSMCs; macrophages; n/a