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Palabras clave – provistas por la editorial

3CAI; GRIA1; GRIA2; intractable epilepsy; PICK1; protein kinase C; pericytes; mural cells; cytokine; blood-brain barrier; neuroinflammation; 3-phosphoinositide-dependent protein kinase-1; AKT; cyclosporin A; GluA1; GluR1; PDK1; refractory seizure; temporal lobe epilepsy; hippocampus; 4-aminopyridine; epilepsy model; long-term potentiation; AMPA receptor; audiogenic epilepsy; rodents; fear reaction; behavior genetics; c-Fos; early gene activation; epileptogenesis; lipopolysaccharide; pentylenetetrazole; post-traumatic epilepsy; seizure susceptibility; traumatic brain injury; epilepsy; ghrelin; ghrelin receptor; anxiety; TRPV1 channels; neuronal activity; neuropsychiatric comorbidity; immunofluorescence; audiogenic kindling; acoustically evoked seizures; EACT; generalized tonic-clonic seizures; inherited epilepsy; TMEM1A channels; wild running seizures; astrocyte; ML365; pharmacoresistant epilepsy; cortical atrophy; hippocampal atrophy; intracerebral electrode; magnetic resonance imaging; posttraumatic epilepsy; n/a