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Palabras clave – provistas por la editorial

microtubule acetylation; triple-negative breast cancer; anti-cancer agent; apoptosis; K562; 8-hydroxydaidzein; autophagy; BCR-ABL; MAPK; NF-κB; c-myc; RNA interference; siRNA; oncogene; gene silencing; expression; nanosystems; Cytochrome P450; CYP1A1; CYP1B1; CYP2W1; breast cancer; prodrug; bioprecursor; duocarmycin; phortress; AQ4N; 5-FU resistance; colorectal cancer; drug repurposing; Genomics of Drug Sensitivity in Cancer; Connectivity Map; anticancer drug; B-lactam steroid alkylators; synthetic lethality; poly (ADP-ribose) polymerase inhibitors; ovarian cancer; hybrid steroidal alkylating agents; migration; invasion; glioblastoma; CCN1; mesenchymal–amoeboid transition; biomarker; HepG2; Huh7; isatin sulfonamides; angiogenesis; cancer hallmarks; molecular docking; EGFR tyrosine kinase inhibitor; photon upconversion; triplet-triplet annihilation; in vivo imaging; PLGA; nanoparticles; affibody molecule; human epidermal growth factor receptor 3 (HER3); BxPC-3; emtansine; DM1; albumin binding domain; affibody drug conjugate (AffiDC); n/a