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Palabras clave – provistas por la editorial

nucleus accumbens-associated protein 1 (NAC1); BEN (BANP, E5R and NAC1) domain; sequence-specific DNA-binding protein; solution NMR structure; isothermal titration calorimetry (ITC); human cytomegalovirus; ovarian cancer; cancer progression; inflammation; immunosuppression; endometrial carcinoma; immune micro-environment; immune checkpoints inhibitors; microsatellite instability; mismatch repair deficiency; platelet-activating factor acetylhydrolase (PAF-AH; PLA2G7); BRCA1 mutant ovarian cancer; Wnt signaling; pGSK3β; β-catenin; prognosis; vulvar melanoma; vaginal melanoma; targeted therapy; gynecological cancer; melanoma treatment; gene ontology; epithelial ovarian cancers; borderline ovarian tumors; differentially expressed genes; aryl hydrocarbon receptor; epithelial–mesenchymal transition; integrative analysis; testin; p16 protein; cervical cancer; cervical neoplasia; immunohistochemistry; chimeric antigen receptor; CD44; adoptive immunotherapy; NK cells; epithelial ovarian cancer; DNA damage response; somatic mutation; clear cell carcinoma; endometrioid endometrial cancer; DNA mismatch repair (MMR); MMR deficient (dMMR); long-term survival; exosome; miRNA; biomarkers; liquid biopsy; interventional radiotherapy; vaginal-cuff brachytherapy; HDR brachytherapy; in vivo dosimetry; endometrial cancer; biological planning; drug repurposing; non-coding RNAs; nanocarriers; anti-angiogenic therapy; n/a