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Palabras clave – provistas por la editorial

MMP; MMP2; MMP9; MMP7; MMP14; matrix metalloproteases; PDAC; pancreatic cancer; Bowman–Birk inhibitor; ranacyclin; trypsin inhibitor; structure–activity relationship; synergistic effect; Gentamicin; matrix metalloproteinase; extracellular matrix; nuclei; cancer; apoptosis; immune response; cysteine protease inhibitor; stefin; signal peptide; parasite; phylogenetic analysis; diversification; protein structure; vascular endothelial growth factors (VEGFs); VEGF-A; PlGF; VEGF-B; VEGF-C; VEGF-D; angiogenesis; lymphangiogenesis; CCBE1; proteases; ADAMTS3; plasmin; cathepsin D; KLK3; prostate-specific antigen (PSA); thrombin; wound healing; metastasis; proteolytic activation; vascular biology; lymphedema