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Palabras clave – provistas por la editorial

androgen receptor; docetaxel; cabazitaxel; castration-resistant prostate cancer; chemotherapy; P-glycoprotein; EPI-002; splice variant; prostate-specific antigen; androgen deprivation therapy; time to PSA nadir; fibroblasts; prostate cancer; androgen sensitivity; pirfenidone; TGFβ1; G1 cell cycle arrest; fibroblast growth factor; fibroblast growth factor receptor; obesity; inflammation; immune cells; cytokine; high-fat diet; KIFC1; docetaxel resistance; apoptosis; CW069; Caveolin-1; TP53-regulated inhibitor of apoptosis 1; tumour stroma; tumour microenvironment; fibroblast; CAF; resistance; radiotherapy; CCL2; CCL22; CCL5; migration; LSD1; epigenetics; autophagy; abiraterone; enzalutamide; testosterone; castration resistant prostate cancer; animal model; diet; fat; in vitro; in vivo; mouse; AKR1C3; hormone-naïve prostate cancer; immunohistochemistry; tissue microarray; androgen receptor dependency; fibroblast-dependent androgen receptor activation; n/a