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Palabras clave – provistas por la editorial

global cerebral ischemia; amiloride; sodium–hydrogen exchanger-1; zinc; neuronal death; neuroprotection; neurodegenerative disorder; choline acetyltransferase (ChAT); trimethyltin (TMT); bean phosphatidylserine (Bean-PS); brain-derived neurotrophic factor; moderate hypoxia; physical exercise; psychomotor function; reaction time; cortisol; catecholamines; nitrite; endotheline-1; lactate; pyridoxine deficiency; ischemia; gerbil; homocysteine; cell death; glia; neurogenesis; N-acetyl-l-cysteine; transient receptor potential melastatin 2; neurodegeneration; Alzheimer’s disease; metabolic disease; adiponectin; insulin; antioxidants; stroke; preventive gene therapy; adenoviral vector; VEGF; GDNF; NCAM; human umbilical cord blood mononuclear cells; antioxidant; brain; neurodegenerative disease; oxidative stress; PGC-1α; vascular endothelial growth factor; vascular endothelial growth factor receptor 2; PI3K/AKT; MEK/ERK; status epilepticus; hippocampus; middle cerebral artery occlusion; reperfusion injury; lipid emulsion; excitotoxicity; apoptosis; GPR4 receptor; MPP+; Parkinson’s disease; CRISPR/cas9; ischemic stroke; blood brain barrier; nanoparticle-based drug delivery; brain targeting; BDNF; miRNAs; synaptic plasticity; depression; glioblastoma; astrocytes; astrocytic networks; connexin 43; calcium activity; neural injury; nimodipine; subarachnoid haemorrhage; acid-sensing ion channels; oxygen-glucose deprivation; liver growth factor; inflammation; microglia; Tg2576 transgenic mice; amyloid-beta; oculomotor system; trophic factors; motoneurons; axotomy; amyotrophic lateral sclerosis; electroneutral transport; cation-chloride cotransporters; KCCs; NKCCs; WNK-SPAK/OSR1; ascorbic acid; aging; organotypic hippocampal slice culture; n/a